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These studies will advance our understanding of the signaling pathways involved in the pathogenesis of NEC and test the novel preventative nutritional strategy of manipulating anti-inflammatory cytokine production with dietary AhR ligands. Name Washington University. Importantly, these findings highlight the therapeutic potential of enhancing IL production to attenuate pro- inflammatory responses seen in NEC. Necrotizing enterocolitis NEC is associated with significant infant mortality and the only effective preventative strategy against NEC is the administration of breast milk. We have shown that breast milk mediates protection against NEC by inhibiting the pro-inflammatory signaling in the intestinal epithelium and we seek to determine the components of breast milk that mediate this anti-inflammatory effect. Our goal is to apply this knowledge to the design of novel preventative formulas for infants at high risk for NEC development.

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Giavanna. Age: 20.
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To test this hypothesis, we will use our experimental NEC model and an ex vivo model of intestinal stem cell isolation and culture to pursue the following specific aims: Aim 1 : To investigate the role of IL producing cells in NEC pathogenesis.

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Paislee. Age: 24.
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Our goal is to apply this knowledge to the design of novel preventative formulas for infants at high risk for NEC development. The current proposal seeks to identify nutritional strategies to protect against NEC and determine the mechanisms involved in mediating the protection. The goals of the proposed research are to identify the innate host defense responses in NEC pathogenesis and interrogate how these responses can be modified or prevented through dietary modifications.

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